Through its action on dopamine (a chemical messenger used between nerve cells) and dopamine receptors, ALC seems to play a major role in preventing and/or minimizing the symptoms of Parkinson’s disease. ALC retards the decline in the number of dopamine receptors that occurs as part of the normal aging process and (more rapidly) with the onset of Parkinson’s disease. In fact, many researchers believe that Parkinson’s may be caused by a deficiency of dopamine. ALC also inhibits tremors and may even play a role in helping with MS by inhibiting (and possibly reversing) the degeneration of myelin sheaths.
Overall, ALC helps slow down the aging process of the brain:
- Retards the inevitable decline in glucocorticoid receptors that occurs with aging.
- Slows the age-related deterioration of the hippocampus.
- Retards the inevitable decline in nerve growth factor receptors that occurs as we age.
- Stimulates and maintains the growth of new neurons within the brain and helps to prevent the death of existing neurons.
- Protects the NMDA (N-methyl-D-aspartic acid) receptors in the brain from age-related decline. NMDA receptors play a critical role in synaptic plasticity, a key cellular mechanism involved in the brain’s learning and memory functions.
- Inhibits the excessive release of adrenaline and the depletion of luteinizing hormone-releasing hormone and testosterone that occurs in response to stress.
- Enhances the function of cytochrome oxidase, an essential enzyme of the electron transport system. This is important because reduced cytochrome oxidase activity is characteristic of neurodegeneration.
The mind-boosting effect of acetyl-L-carnitine is often noticed within a few hours, even within an hour, of supplementing. Most people report feeling mentally sharper, having more focus, and being more alert. And ACL, like DMAE, helps flush lipofuscin from the body, especially from the brain."
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