"There’s an ad on TV with a message something like, 'Cholesterol comes from the tiramisu you had for dessert and from your Aunt Sue. If changing your diet isn’t enough, be sure and ask your doctor about our drug.' That ad totally misses the point. In fact, as I pointed out earlier, diet only accounts for about 20 percent of your cholesterol level. Certainly, genetics matters if it short-changed you on the number of liver LDL receptor sites you have. But that hardly limits your options to statin drugs. You always have the option of optimizing the functionality of the receptor sites that you do have in order to facilitate the movement of cholesterol into the liver, out through the bile duct, and then out through the colon. How?
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- Avoid trans-fatty acids like the plague -- Hydrogenated and partially hydrogenated oils (the trans-fatty acids) are the number one killer in the modern diet. Among other things, they cause the LDL receptor sites to overload.
- Optimize the liver -- Do a periodic liver flush that includes the use of lipotropic herbs (herbs that promote the utilization of fats), such as dandelion root, to flush accumulated fats and cholesterol from the liver and gallbladder. By flushing fats from the liver, you facilitate the movement of cholesterol from the surface LDL sites into the liver and then on out through the bile duct.
- Supplement with water-soluble fiber such as psyllium or oat bran -- Water soluble fiber lowers total cholesterol and LDL cholesterol by binding with bile acids and preventing their re-absorption into the body. This lowers cholesterol as cholesterol is a major component of bile acids, which are used by the body to aid in the digestion of fats. If soluble fiber is present in the intestinal tract, it binds with the bile acids after they have helped break down the fats in your diet and escorts them out of the body. Since they are not reabsorbed because of the presence of water-soluble fiber, the liver has to draw more cholesterol from the blood to make more bile acids to be used for further digestion. The net effect is to lower cholesterol levels in the blood.
- Lower homocysteine levels -- While there is a considerable amount we do not know about homocysteine, we do know how to use nutritional supplements to reduce its levels. This is done using a combination of folic acid, vitamin B12, and trimethylglycine (TMG), which facilitates a process called methylation, along with vitamin B6, which facilitates trans-sulfuration. Such a combined approach can normalize homocysteine in 95 percent of people.
- Optimize omega-6 to omega-3 fatty acid ratios -- Begin by eliminating your use of the bottled vegetable oils found in your supermarket, except for olive oil or walnut oil, and supplementing with fish oil, krill oil, perilla oil, or flaxseed oil, which are all high in omega-3s. Much of the problem with inflammatory disorders actually stems from a lopsided imbalance in dietary intake of the omega-6 and omega-3 fatty acids and the resulting cascade in pro-inflammatory activity. The ideal ratio is roughly 1 to 1; however, as we discussed previously, people in industrialized countries have replaced much of their dietary saturated fat (on the mistaken advice of their doctors and the media) with vegetable oil omega-6s. Ratios of 20 to 1 and 30 to 1 are now not uncommon. From a biochemical standpoint, this sets the stage for major arterial inflammation.
- Take antioxidants -- A good antioxidant formula that contains oligomeric proanthocyanidins (OPCs), such as green tea, pine bark, and grape seed extract, can help repair damage to arterial walls (see Chapter 10).
- Use proteolytic enzymes -- This is one of the most important things you can do. The regular use of proteolytic enzymes can help eliminate CICs from the body, reduce overall inflammation, dissolve accumulated plaque, and repair arterial scar tissue. Although the evidence is purely anecdotal at the moment, we have seen extraordinary results using detoxification levels of these enzymes (see Chapter 11)."
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